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Cause of neurological Covid-19 symptoms explored

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CLINICIANS have found that Covid-19 is associated with neurological symptoms.
Scientists need to conduct more research to understand how Covid-19 is linked to these neurological symptoms.

Researchers have presented their findings on these links at Neuroscience 2021, the annual conference of the Society for Neuroscience.

Researchers have been exploring the way Covid-19 affects a person’s nervous system, causing neurological symptoms.

This week, they presented their findings at Neuroscience 2021. This event is the annual meeting of the Society for Neuroscience, which is the largest global society for scientists and clinicians focused on issues of brain and nervous system health.

The findings indicate how SARS-CoV-2 — the virus that causes Covid-19 — gains access to a person’s brain. They also provide insight into the effects it has once it has entered this part of the body.

For most people, the primary symptoms of Covid-19 are respiratory. According to the World Health Organization (WHO)Trusted Source, the most common symptoms of Covid-19 are fever, a cough, tiredness, and the loss of taste or smell.

These symptoms and other, less common ones can range in severity from mild to life threatening. Some people who acquire a SARS-CoV-2 infection may even remain asymptomatic.

As well as these respiratory symptoms, clinicians and researchers have also observed various neurological symptoms associated with Covid-19.

Researchers have linked Covid-19 to headaches, brain inflammation, muscle weakness or pain, and numbness or weakness in a person’s hands, feet, or limbs.

However, further research is necessary to understand how Covid-19 affects a person’s nervous system.

Dr. Ashutosh Kumar, an assistant professor in the Department of Anatomy at the All India Institute of Medical Sciences, Patna, India, presented findings that identified a possible receptor that may give SARS-CoV-2 access to a person’s brain.

In other parts of the body, SARS-CoV-2 gains access to cells by binding to the protein angiotensin converting enzyme 2 (ACE2) with the help of transmembrane serine protease 2 (TMPRSS2).

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